Diabetic foot ulcers are slow-healing and increase the risk of infections and amputation. Researchers at Karolinska Institutet have identified a circular RNA in mitochondria, circMTRNR2, that plays an important role in the healing of chronic wounds. The RNA was found to be reduced in tissue from patients with long-standing diabetic foot ulcers. The molecule appears to support the skin’s reparative capacity by protecting the cells’ energy metabolism against harmful stress. The study is published in Advanced Science.
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